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Further, after 1980 and 2000 data were compared, it was estimated that approximately 47% of the decrease in CHD deaths was attributable to treatments, including secondary preventive therapies, initial treatment after acute myocardial infarction, and revascularization for chronic angina.
Ranolazine may cause some small changes in laboratory parameters such as an increase in eosinophil count, an increase in creatinine, a decrease in hematocrit, and a decrease in hemoglobin A 1C in diabetic patients.
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We observed that 3 mo of metoprolol treatment in dogs with coronary microembolization-induced heart failure improved LV function and a significant 28% decrease in CPT-I activity (), suggesting that the shift in substrate metabolism observed in the clinical studies could be due to downregulation of this enzyme.
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One death occurred during the study, 92 days after the last dose of study drug.
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In a previous trial, antianginal monotherapy with ranolazine, a drug believed to partially inhibit fatty acid oxidation, increased treadmill exercise performance; however, its long-term efficacy and safety have not been studied in combination with beta-blockers or calcium antagonists in a large patient population with severe chronic angina.
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ICA should not be performed in patients with angina who refuse invasive procedures, prefer to avoid revascularization, who are not candidates for PCI or CABG, or in whom revascularization is not expected to improve functional status or quality of life.
Treatment of isolated working rat hearts with leptin also increases fatty acid β-oxidation of both exogenously and endogenously derived fatty acids, and is associated with a decrease in intramyocardial TAG stores ().
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